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imaging software nis elements version 3 0 br  (Nikon)


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    Structured Review

    Nikon imaging software nis elements version 3 0 br
    Imaging Software Nis Elements Version 3 0 Br, supplied by Nikon, used in various techniques. Bioz Stars score: 99/100, based on 39904 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/imaging+software+nis+elements+version+3+0+br/NIS-Elements/pm41577290-92-16-23
    Average 99 stars, based on 39904 article reviews
    imaging software nis elements version 3 0 br - by Bioz Stars, 2026-10
    99/100 stars

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    Related Articles

    Microscopy:

    Article Title: Leptin Increases Lipid Storage in Sertoli Cells: Signaling Pathways and Cellular Mechanisms Involved.
    Article Snippet: Leptin is an adipokine with a role in male reproductive function.. Sertoli cells (SCs) express leptin receptors, but leptin effects on SCs’ functions have been scarcely researched.. SCs support germ cell development by providing various compounds, including transferrin and ketone bodies.

    Imaging:

    Article Title: Leptin Increases Lipid Storage in Sertoli Cells: Signaling Pathways and Cellular Mechanisms Involved.
    Article Snippet: Leptin is an adipokine with a role in male reproductive function.. Sertoli cells (SCs) express leptin receptors, but leptin effects on SCs’ functions have been scarcely researched.. SCs support germ cell development by providing various compounds, including transferrin and ketone bodies.

    Software:

    Article Title: Leptin Increases Lipid Storage in Sertoli Cells: Signaling Pathways and Cellular Mechanisms Involved.
    Article Snippet: Leptin is an adipokine with a role in male reproductive function.. Sertoli cells (SCs) express leptin receptors, but leptin effects on SCs’ functions have been scarcely researched.. SCs support germ cell development by providing various compounds, including transferrin and ketone bodies.



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    Nikon imaging software nis elements br version 3 0
    Figure 1. TNF‑α induces VSMC apoptosis. (A) Murine VSMCs were stimulated with 0‑50 ng/ml TNF‑α for 24 h. Cell death was measured using a Cell Death Detection ELISA kit and is expressed as ELISA absorbance units. (B) VSMC apoptosis was determined using the TUNEL assay, following stimulation with 10 ng/ml TNF‑α for 24 h. White arrows indicate apoptotic VSMCs (magnification, <t>x100).</t> (C) Quantitative analysis of TUNEL results. n=5. *P<0.05 vs. 0 ng/ml TNF‑α. TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucleotidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.
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    Figure 1. TNF‑α induces VSMC apoptosis. (A) Murine VSMCs were stimulated with 0‑50 ng/ml TNF‑α for 24 h. Cell death was measured using a Cell Death Detection ELISA kit and is expressed as ELISA absorbance units. (B) VSMC apoptosis was determined using the TUNEL assay, following stimulation with 10 ng/ml TNF‑α for 24 h. White arrows indicate apoptotic VSMCs (magnification, <t>x100).</t> (C) Quantitative analysis of TUNEL results. n=5. *P<0.05 vs. 0 ng/ml TNF‑α. TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucleotidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.
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    Image Search Results


    Figure 1. TNF‑α induces VSMC apoptosis. (A) Murine VSMCs were stimulated with 0‑50 ng/ml TNF‑α for 24 h. Cell death was measured using a Cell Death Detection ELISA kit and is expressed as ELISA absorbance units. (B) VSMC apoptosis was determined using the TUNEL assay, following stimulation with 10 ng/ml TNF‑α for 24 h. White arrows indicate apoptotic VSMCs (magnification, x100). (C) Quantitative analysis of TUNEL results. n=5. *P<0.05 vs. 0 ng/ml TNF‑α. TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucleotidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.

    Journal: Molecular medicine reports

    Article Title: MicroRNA‑494 inhibits apoptosis of murine vascular smooth muscle cells in vitro.

    doi: 10.3892/mmr.2019.10085

    Figure Lengend Snippet: Figure 1. TNF‑α induces VSMC apoptosis. (A) Murine VSMCs were stimulated with 0‑50 ng/ml TNF‑α for 24 h. Cell death was measured using a Cell Death Detection ELISA kit and is expressed as ELISA absorbance units. (B) VSMC apoptosis was determined using the TUNEL assay, following stimulation with 10 ng/ml TNF‑α for 24 h. White arrows indicate apoptotic VSMCs (magnification, x100). (C) Quantitative analysis of TUNEL results. n=5. *P<0.05 vs. 0 ng/ml TNF‑α. TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucleotidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.

    Article Snippet: Images were processed using Imaging Software NIS-Elements BR version 3.0 (Nikon Corporation; magnification, x100).

    Techniques: Enzyme-linked Immunosorbent Assay, TUNEL Assay, End Labeling

    Figure 6. miR‑494 inhibits TNF‑α induced VSMC apoptosis via BCL2L11. (A) Efficiency of siRNA knockdown of BCL2L11 in murine VSMCs was determined by western blot analysis. Murine VSMCs were transfected with scramble siRNA + control miR inhibitors, scramble siRNA + miR‑494 inhibitors, BCL2L11 siRNA + control miR inhibitors and BCL2L11 siRNA + miR‑494 inhibitor. Subsequently, 48 h post‑transfection, cells were incubated with 10 ng/ml TNF‑α for 24 h. (B) Cell apoptosis was measured by TUNEL assay (white arrows indicate apoptotic VSMCs; magnification, x100). (C) Quantitative analysis of TUNEL results. (D) Cell death ELISA was performed and cell apoptosis is presented as ELISA absorbance units. n=5. *P<0.05, **P<0.01, ***P<0.001. BCL2L11, B‑cell lymphoma-2‑like 11; miR, microRNA; NC, negative control; siRNA, small interfering RNA; TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucle otidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.

    Journal: Molecular medicine reports

    Article Title: MicroRNA‑494 inhibits apoptosis of murine vascular smooth muscle cells in vitro.

    doi: 10.3892/mmr.2019.10085

    Figure Lengend Snippet: Figure 6. miR‑494 inhibits TNF‑α induced VSMC apoptosis via BCL2L11. (A) Efficiency of siRNA knockdown of BCL2L11 in murine VSMCs was determined by western blot analysis. Murine VSMCs were transfected with scramble siRNA + control miR inhibitors, scramble siRNA + miR‑494 inhibitors, BCL2L11 siRNA + control miR inhibitors and BCL2L11 siRNA + miR‑494 inhibitor. Subsequently, 48 h post‑transfection, cells were incubated with 10 ng/ml TNF‑α for 24 h. (B) Cell apoptosis was measured by TUNEL assay (white arrows indicate apoptotic VSMCs; magnification, x100). (C) Quantitative analysis of TUNEL results. (D) Cell death ELISA was performed and cell apoptosis is presented as ELISA absorbance units. n=5. *P<0.05, **P<0.01, ***P<0.001. BCL2L11, B‑cell lymphoma-2‑like 11; miR, microRNA; NC, negative control; siRNA, small interfering RNA; TNF‑α, tumor necrosis factor‑α; TUNEL, terminal deoxynucle otidyl‑transferase‑mediated dUTP nick end labeling; VSMC, vascular smooth muscle cell.

    Article Snippet: Images were processed using Imaging Software NIS-Elements BR version 3.0 (Nikon Corporation; magnification, x100).

    Techniques: Knockdown, Western Blot, Transfection, Control, Incubation, TUNEL Assay, Enzyme-linked Immunosorbent Assay, Negative Control, Small Interfering RNA, End Labeling